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賽立復(fù)NMN除了抗衰,對(duì)人體還有哪些好處?

作者:上海大器租車公司???時(shí)間:2021-01-24 09:10

賽立復(fù)NMN都是經(jīng)過(guò)純化后的β-NMN

賽立復(fù)年青素(NMN)安全檢測(cè)報(bào)告書

現(xiàn)今,每月都不斷有新的NMN品牌涌現(xiàn),NMN也收到資本追捧。但對(duì)消費(fèi)者來(lái)說(shuō),最重要的還是選擇安全可靠的產(chǎn)品。

引用文獻(xiàn):

【1】Mills K , Yoshida S , Stein L , et al. Long-Term Administration of Nicotin**ide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice[J]. Cell Metabolism, 2016, 24(6):795-806. (2016年)

【2】Nicotin**ide mononucleotide, a key NAD(+) intermediate, treats the pathophysiology of ***t- and age-induced diabetes in mice. (2011年)

【3】Nicotin**ide mononucleotide protects against pro-infl****tory cytokine-mediated impairment of mouse islet function. (2011年)

【4】 Nicotin**ide mononucleotide inhibits JNK activation to reverse Alzheimer disease. (2017年)

【5】 NAD replenishment with nicotin**ide mononucleotide protects blood-brain barrier integrity and attenuates delayed tissue plasminogen activator-induced haemorrhagic transfor**tion after cerebral ischaemia. (2017年)

【6】 Effect of nicotin**ide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer’s disease-relevant murine model (2015年)

【7】Nicotin**ide mononucleotide protects against β-**yloid oligomer-induced cognitive impairment and neuronal death. (2016年)

【8】Long-term administration of nicotin**ide mononucleotide mitigates age-associated physiological decline in mice (2016年)

【9】Bonkowski M S , Das A , Schultz M B , et al. IMPAIRMENT OF AN ENDOTHELIAL NAD+-H2S SIGNALING NETWORK IS A REVERSIBLE CAUSE OF VASCULAR AGING[J]. Innovation in Aging(suppl_1):suppl_1.

【10】Mills K , Yoshida S , Stein L , et al. Long-Term Administration of Nicotin**ide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice[J]. Cell Metabolism, 2016, 24(6):795-806.